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Blood Interpretation - Fasting Glucose
Article Index
Blood Interpretation
HDL Cholesterol
LDL Cholesterol
Lipoprotein a (Lp(a))
Apolipoprotein A-l (Apo A-1)
Apolipoprotein B (Apo B)
C-Reactive Protein
Fasting Glucose
Fasting Serum Insulin
Carbon Dioxide (CO2)
BUN (Blood Urea Nitrogen)
BUN/Creatinine Ratio
Uric Acid
Alkaline Phosphatase
Total Protein
Albumin/Globulin Ratio: (A/G Ratio)
Calcium/Albumin Ratio: (Ca/A ratio)
GGT (Gamma-glutamyl transferase)
SGOT: (also known as AST)
SGPT: (also known as ALT)
Total Bilirubin
Serum Iron (Fe)
Thyroid Profile
T4 : (tetra-iodothyronine)
T7: (also known as FTI [free thyroid index])
Basal Body Temperatute Studies for Thyroid Function
All Pages

Fasting Glucose


(normal range is 80 - 100; ideal range is 65-85)

Glucose is very acidic. This is why diabetics have a tendency towards acidosis. The whole system surrounding how our bodies utilize glucose as well as the organs that participate in that utilization, is quite involved.


If glucose is elevated, consider the following:

  • Diabetes. Be sure to run a fasting serum glucose and a Hemoglobin A-1C (protein bound glucose), and a simple urinalysis (glucosuria usually does not occur until plasma value reaches 180)
  • Along with elevated cholesterol (>220) and triglycerides (150) suspect hyperinsulinemia (syndromeX)
  • Along with decreased CO2 (<25) and an increased anion gap (>12), suspect thiamine (B1) deficiency

Other disease processes/conditions that may elevate glucose include: infections (if WBC count is >18,000), chronic renal disease, hyperthyroid, hyper function of the adrenals (Cushing's disease), MI, occasionally pregnancy, inflammatory bowel conditions, asthma, pancreatitis, brain trauma, severe trauma of any sort, convulsions, severe liver disease, and the first 24 hours after a severe burn.

Drugs that may also cause one's serum glucose levels to elevate include: ACTH, corticosteroids, epinephrine,

furosamide, thiazides, phenytoin.


The following nutritional agents may be considered for those with an elevated serum glucose:

  • Modify the diet. Diets low in calories high in fiber but low in simple carbohydrates and saturated or trans-

fatty acids may help. (FirstLine® Therapy Diet)

  • Exercise: The uptake of glucose into muscle is a significant means of glucose disposal and takes place

independently from the action of insulin.

  • B complex, including B6 to convert tryptophan to niacin and for gluconeogenesis), B1 (for conversion of pyruvic acid to acetyl CoA), B12 (for methylmalonate conversion to succinate for carbohydrate and fat metabolism), Niacin (B3 to make GTF and NAD), magnesium, zinc (for insulin synthesis by pancreatic B cells and to address the post-receptor defect), chromium (to make glucose tolerance factor and address the pre-receptor defect), copper (for insulin binding), biotin (needed for glucose utilization in the cells), vanadium (has possible insulintropic effects)

UltraGlycemX - follow modified step program in Blood Sugar Section

MetaGlycemX - 1-2 tablets twice daily

  • Botanicals including: gymnema, trigonella (fenugreek seeds), momordica (bitter melon), ficus (fig leaf)

Fenugreek Plus - 2 tablets daily in-between meals


If glucose is low, consider the following:

  • Hypoglycemia (reactive). 60% of the population is hypoglycemic and the majority of those individuals will possess food allergies, particularly to the grains. Not uncommonly, the LDH will be decreased (below

150). LDH activity represents the active exchange of chloride with glucose and glucose with zinc and

sodium (glycolysis), therefore, LDH activity is associated with pancreatic function and glucose metabolism.

  • Many nutritional medicine IV infusions can cause a reactive hypoglycemic event. Follow protocol.
  • Insulinoma: Tumors should be ruled out with unexplained decreased fasting glucose. An insulin/glucose

ratio of greater than .03 is presumptive of insulinoma.

  • Possible sprue or celiac disease especially with a flat curve on GTT (25mg/100ml) although a flat curve

on a GTT has also been seen with hypothyroidism and pituitary insufficiency, addison's disease (with a concurrent decreased sodium and increased potassium)

Other conditions may cause a low, fasting glucose including: Protein malnutrition, occasionally pregnancy, hypoadrenia, hypochlorhydria, and liver disease (destruction or insufficiency), certain types of heavy metal burdens.

Several medications may also cause low blood glucose levels including: Acetohexamine allopurinol, aminosalicylic acid, amodiaquine amphotericin B, steroids, androgens, choorpropamide, cyclophosphamide, desipramine, erythroycin, glycopyrrolate, haloperidol, halothane, hydrazine, imipramine, indomethacin, isoniazid, lincomycin, MAO inhibitors, mercaptopurine, metaxalone, methoxsalen, methoxyflurane, methyldopa, methly-thiouracil, nicotinic acid, nitrofurantoin, novobiocin, oleandomycin, oxazepam, oxyphenbutazone, paraldehyde, paramethadione, phenacemide, phenacetin, phenothiazines, phenybutazone, progestins and estrogens, propranolol, propylthiouracil, quinacrine, sulfonamides, tetracyclines thiosemicarbazones thiothixene, tolazamide, trimethadione, uracil.

The following nutritional agents may be considered for those who exhibit low serum

glucose levels:

  • Diet. Eat numerous small meals throughout the day. Be aware that hypochlorhydria, biliary stasis and hepatic insufficiency are common with glycemic problems (both high and low). A diet high in protein may

be contraindicated. Refined carbs should be stopped, and fresh, raw fruits such as berries and leafy

green vegetables should be a staple. If digestive problems are present, limit lean meat to 4 to 6 ounces per day.

  • The same vitamins and minerals and herbals as listed above with elevated glucose values
  • Address the underlying clinical condition that may be contributing
  • Address the underlying drug therapy that may be influencing the low blood sugar status.


Clinical observations:

One may need to run a Reinch test (hair mineral analysis for toxic, heavy metals). Some studies implicate heavy

metals has a contributing factor in dysglycemic conditions.

Fasting blood glucose is generally able to identify (initial) hyperglycemic conditions, although in hypoglycemia, the blood glucose is often not below homeostatic ranges. A 5 to 6 hour glucose tolerance test (GTT) can be performed to identify reactive hypoglycemia. One must note that within that test that can be quite symptomatic

for the patient being tested, that a GTT value of 15 points or more below the fasting level is indicative of reactive


Those diabetic patients (Type I - insulin dependant or Type II) need to be monitored with a test called glycohemoglobin

A-1C. This test will tell us what the patient's blood sugar levels are averaging over a period of 4 to 8 weeks.

Last Updated on Saturday, 14 March 2009 02:39